TRABID inhibition activates cGAS/STING-mediated anti-tumor
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IBC - 最新發表論文: TRABID inhibition activates cGAS/STING-mediated anti-tumor immunity through mitosis and autophagy dysregulation
VPS34 K29/K48 branched ubiquitination governed by UBE3C and TRABID regulates autophagy, proteostasis and liver metabolism
Mitotic SENP3 activation couples with cGAS signaling in tumor cells to stimulate anti-tumor immunity
TRABID inhibition activates cGAS/STING-mediated anti-tumor immunity through mitosis and autophagy dysregulation
TRAF3 activates STING-mediated suppression of EV-A71 and target of viral evasion
The antitumor and protumor effects of cGAS-STING activation. ( A and B
Characterization of TRABID Specificity
TRAF4 regulates ubiquitination-modulated survivin turnover and confers radioresistance
cGAS-STING pathway in cancer biotherapy, Molecular Cancer
TRABID在抑制抗肿瘤免疫中的关键作用- 微生物安全与健康网
TRABID inhibition activates cGAS/STING-mediated anti-tumor immunity through mitosis and autophagy dysregulation
The Buzz Around STING Agonists
LIR domain of CGAS was required for CGAS-LC3 interaction. (A)
Cancers, Free Full-Text
Mitotic SENP3 activation couples with cGAS signaling in tumor cells to stimulate anti-tumor immunity
